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The pathological consequences of interaction between environmental carbon pollutants and microbial antigens have not been fully explored. We developed a murine model of multi-wall carbon nanotube MWCNT -elicited granulomatous disease which bears a striking resemblance to sarcoidosis, a human granulomatous disease. Because of reports describing lymphocyte reactivity to mycobacterial antigens in sarcoidosis patients, we hypothesized that addition of mycobacterial antigen ESAT-6 to MWCNT might elicit activation in T cells. Determining the potentially adverse effects of environmental carbon pollutants on human health is a difficult and continuing problem. Production and usage of engineered nanomaterials represent a growing technology [ 1 ] while combustion-generated carbon pollution is ubiquitous and can include both carbon nanotubes and nanoparticles [ 2 ].
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Background: The pathological consequences of interaction between environmental carbon pollutants and microbial antigens have not been fully explored. We developed a murine model of multi-wall carbon nanotube MWCNT - elicited granulomatous disease which bears a striking resemblance to sarcoidosis, a human granulomatous disease. Because of reports describing lymphocyte reactivity to mycobacterial antigens in sarcoidosis patients, we hypothesized that addition of mycobacterial antigen ESAT-6 to MWCNT might elicit activation in T cells. Determining the potentially adverse effects of environmental carbon pollutants on human health is a difficult and continuing problem. Production and usage of engineered nanomaterials represent a growing technology [ 1 ] while combustion-generated carbon pollution is ubiquitous and can include both carbon nanotubes and nanoparticles [ 2 ]. Animal model studies suggest that multiwall carbon nanotubes MWCNT can affect immunological responses, produce granulomas, or elicit fibrosis, depending upon chemical and structural characteristics [ 3 , 4 ].
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